2.79J/2.79J/3.96J/BE.441J/HST.522J
A theory of induced
regeneration in adults.
Note: [C, S, R] data only cited;
no kinetics
Outline
1. Irreversible injury
2. Regenerative and nonregenerative
tissues
3. Antagonistic relation between
contraction and regeneration
4. Present theory: Selective inhibition of
contraction necessary but not sufficient
for regeneration
5. Mechanism
1. Irreversible injury
Reversible injury
Image removed due to copyright considerations.
See Figure 1.1 in Yannas, I. V. Tissue and Organ
Regeneration in Adults. New York: Springer-Verlag, 2001.
Image removed due to copyright consi erations.
See Figure 1.1 in Yannas, I. V. Tissue and Organ
Regeneration i Adults. New York: Springer-Verlag, 2001.
Spontaneous regeneration of amputated limb in the
newt occurs independently of severity of injury
Goss, 1992
Irreversible injury
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Burn victim suffering
from severe contraction
and scar formation
Tomasek et al., 2000
2. Regenerative and
nonregenerative tissues
in adult mammals
The tissue triad in skin and
nerves
Skin
The epidermis is a regenerative tissue. After
excision, it regenerates spontaneously.
Reversible injury. No contraction.
The dermis is a nonregenerative tissue in the
adult. After excision, it does not regenerate
spontaneously. Irreversible injury.
Contraction occurs with scar formation.
Skin
Peripheral nerve
The myelin sheath is a regenerative tissue.
Following nerve crushing with myelin
disruption, the myelin regenerates
spontaneously. Reversible injury. No
contraction.
crushed nerve
heals
spontaneously
by
regeneration
Peripheral nerve
The endoneurial stroma is a nonregenerative
tissue.Following transection, it forms neural
scar (neuroma). Irreversible injury.
Contraction occurs.
transected
nerve heals
spontaneously
by contraction
and neuroma
(neural scar)
formation
SUMMARY SO FAR
Regenerative
tissues.
Reversible injury.
No contraction.
Nonregenerative
tissues. Irrever-
sible injury.
Contraction
+scar.
SKIN epidermis dermis
BM
NERVE myelin endoneurial
stroma
BM
Conclusion: Inverse relation
between contraction and
regeneration
? During adult healing, no contraction
is observed following injury to
epithelia or basement membrane.
? Contraction is only observed
following injury to stroma.
? Contraction only observed following
irreversible injury.
3. Antagonistic relation
between contraction and
regeneration
a. Data from spontaneously healing
wounds
b. Blocking of contraction using scaffolds
c. Isolation of contraction during “island”
grafting
d. Scar formation vs inhibition of
contraction
e. Contraction during impaired healing
Quantitative description of
healing processes
? Initial wound area is A
o
? Wound eventually closes up
spontaneously. Final area is A
f
.
? Final wound area is distributed among
fractions that closed by contraction (%C),
scar formation (%S) or regeneration (%R).
? This is the configuration of the final state.
? Wound closure rule:
C + S + R = 100
Spontaneously
healing defect
Configuration of
final state
general case [C, S, R]
ideal fetal healing [0, 0, 100]
dermis-free skin--
adult rodents
[96, 4, 0]
dermis-free skin--
adult human
[37, 63, 0]
peripheral nerve–
adult rat
[96, 4, 0]
conjunctiva--
adult rabbit
[45, 55, 0]
a. Data from spontaneously
healing wounds
Tadpole → Frog
Developmental changes in configuration
of final state [C, S, R] following healing:
(early stages) → (late stages)
[41, 0, 59] → [62, 0, 38] → [66, 0, 34] → [90, 10, 0]
tadpole → frog
b. Blocking of contraction using
scaffolds
Organ/
species
Treatment
used
Spontan-
eous
Treated
skin-guinea
pig
scaffold A [91, 9, 0][89, 0, 11]
skin-guinea
pig
scaffold
DRT+ KC
[92, 8, 0][28, 0, 72]
nerve-rat silicone
tube+scaf-
fold DRT
[95, 5, 0][53, 0, 47]
nerve-rat collagen
tube+scaf-
fold NRT
[95, 5, 0][0, 0, 100]
conjunctiva
-rabbit
scaffold DRT [45, 55, 0][13, 0, 87]
Skin
Skin
Burn patient
has closed
severe skin
wounds in
neck partly
by
contraction
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Wound area closure using three protocols
[89, 0, 11]
[28, 0, 72]
[91, 9, 0]
Skin
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See Figure 8.1 in [Yannas].
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See Figure 8.1 in [Yannas].
Dermis regeneration template (DRT)
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Structural features of biologically ECM analogs
3. pore structure (ligand density)
2. macromolecular
structure (ligand
duration)
1. chemical composition (ligand identity)
4. orientation of pore channels (ligand orientation)
Diagrams removed due to copyright considerations.
Contraction
inhibited maximally
in pore
diameter range
20 μm — 120 μm.
Scaffolds with pores
in that range induced
dermis regeneration.
Scar formed outside
that range.
DRT
Image removed due to copyright
considerations.
See Figure 8.5 - top in [Yannas].
ue to copyright
consi erations.
See Figure 8.5 - top i [Yannas].
rete ridges with
capillary loops
and vascular plexus
underneath
Normal skin
Image removed due to copyright considerations.
See Figure 5.2 (top left) in [Yannas].
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See Figure 5.2 (top left) in [Yannas].
Burkitt et al., 1992
Verify induced regeneration of skin basement
membrane.
I: Immunostaining: Factor VIII for capillary loops
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75 μm
Compton et al., 2000
Verify induced regeneration of skin basement
membrane.
II. Immunostaining: α
6
β
4
Integrin for
hemidesmosomes
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100 μm
Compton et al., 2000
Verify induced regeneration of skin basement
membrane.
III. Immunostaining: Collagen VII for anchoring fibrils
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150 μm
Compton et al.,2000
Conjunctiva
Anatomy of the conjunctiva
Fornix
Eyelid
Cornea
Sclera
Epithelium
Substantia
Propria
Conjunctival
stroma
low mag
high mag
Conjunctiva wound model
Conjunctiva
Tenon’s capsule
Sclera
Sclera
Scaffold DRT
Sutures
Effect of DRT on contraction kinetics of conjunctival defect. It is
experimentally convenient to study contraction of the fornix,
a tissue attached to the conjunctiva.
ungrafted
grafted with scaffold DRT
30
% Fornix Shor
tening
15
0
15 300
Days
Hsu et al., 2000
Test of synthesis of conjunctival stroma
(use microscope polarizing stage to study orientation of
collagen fibers)
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ungrafted
conjunctival scar
grafted with scaffold
DRT
normal conjunctiva
Hsu et al., 2000
Peripheral nerve
Rat sciatic nerve model
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Experimental model used to study
PNS regeneration
? Transect rat sciatic nerve
? Insert nerve stumps into tube
? “Nerve chamber” model is standard
proximal
stump
tube, empty
or filled
distal
stump
gap
Nerve
regenera-
ted
across 8-
mm gap
Image removed due to copyright considerations.
See Figure 10.7 (lower right) in [Yannas].
Image removed due to copyright consi erations.
See Figure 10.7 (lower right) in [Yannas].
Jenq and Coggeshall, 1985
Contractile cell zone surrounds regenerating
nerve
contractile cells
original stump surface
regenerated nerve
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Spilker and Seog, 2000
Nerve regeneration template (NRT)
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silicone tube
scaffold tube
Images removed due to copyright considerations.
I r d due t c right c iderations.
15-20 contractile cell layers
poor quality of regeneration
0-1 contractile cell layer
superior quality
Chamberlain et al., 2000
c. Scar formation vs inhibition of
contraction
Data in table will show: Inhibition of
contraction, even modest, leads to
virtual abolition of scar
Scar appears to be a by-product of
contraction
Organ/
species
Treatment
used
Spontan-
eous
Treated
skin-guinea
pig
scaffold A [91, 9, 0][89, 0, 11]
skin-guinea
pig
scaffold
DRT+ KC
[92, 8, 0][28, 0, 72]
nerve-rat silicone
tube+scaf-
fold DRT
[95, 5, 0][53, 0, 47]
nerve-rat collagen
tube+scaf-
fold NRT
[95, 5, 0][0, 0, 100]
conjunctiva
-rabbit
scaffold DRT [45, 55, 0][13, 0, 87]
d. Impaired healing of skin
wounds
Dermis-free wounds in:
? genetically diabetic mouse
? genetically obese mouse
? infected wounds
? mechanically splinted
? treated with steroids
all impaired-healing wounds showed strong
delay in contraction but not regeneration
4. Theory: Selective
inhibition of contraction
Conclusions
1. During development, contraction increases in
importance, while regeneration
corespondingly decreases (rana catesbeiana).
2. Certain scaffolds block contraction
“selectively” and induce partial regeneration
in adult mammals (rodents, swine, human).
3. Scar is abolished when contraction is
blocked.
4. Impaired healing blocks contraction but does
not induce regeneration.
Theory
refers to [C, S, R]
? Inhibition of contraction is necessary
but does not suffice to induce
regeneration
?R > 0 and S → 0 if ?C < 0
Mechanism of contraction
inhibition by DRT scaffold in skin wound
? Scaffold does not aggregate platelets
(during its preparation, abolish collagen
banding but not triple helix). Hypothesis:
Downregulate release of TGF-β.
? Scaffold binds TGF- β1 avidly (but
nonspecifically). Hypothesis: Downregulate
soluble cytokine concentration.
? Scaffold binds myofibroblasts extensively.
Hypothesis: myofibroblast contractile axes
disoriented, lose vectorial character.
? Scaffold competitively inhibits natural ECM
Myofibroblast
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Tomasek et al., 2000
100 μm
100 μm
A. No scaffold.
Contracting wound
B. Grafted with scaffold.
No contraction
Myofibroblasts stain brown-red. Scaffold unstained.
surface of wound
surface of wound
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Kidney
Test applicability of theory to a new
organ
Rat kidney fibrotic tissue
stains blue
untreated scar formation
and contraction of perimeter
significantly smaller scar and
less contraction of perimeter
treated
with
scaffold
DRT
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