Methodology of Organ
Synthesis
A. Skin
B. Conjunctiva
C. Peripheral nerves
D. Simplest synthetic pathways
The tissue triad in skin and
nerves
A. Skin synthesis in vivo or
regeneration (Ch. 5)
1. Experimental parameters
2. Synthesis of epidermis and BM
3. Synthesis of dermis
4. Partial synthesis of skin
5. Comparative regenerative activity
1. Experimental parameters
(skin)
A. Anatomically well-defined defect
–Designate experimental volume
–Delete nonregenerative tissue(s)
–Anatomical bounds
–Containment of exudate
B. Timescale of observations
–Initial state: defect generated
–Final state: defect closed
Note: Remodeling continues after closure
Image removed due to copyright considerations.
See Figure 3.1 in Yannas, I. V. Tissue and Organ
Regeneration in Adults. New York: Springer-Verlag, 2001.
Image removed due to copyright consi erations.
See Figure 3.1 in Yannas, I. V. Tissue and Organ
Regeneration i Adults. New York: Springer-Verlag, 2001.
Standardized reactors
? Transected nerve
SKIN
PERIPHERAL
NERVE
dermis
epidermis
nerve stumps
1. Experimental parameters
(skin) [Cont.]
C. Assays of configuration (final state)
-Literature describes several assays,
unrelated to nature of product synthesized
(e.g., time of closure by epithelialization,
% take of graft, ability to cross
histocompatibility barriers).
-Required assays are both qualitative (which
tissue was synthesized?) and quantitative
(How much?)
Defect closure rule
C + S + R = 100
Assays for C, S and R: see Ch. 4
spontaneous healing of full-thickness
skin excision by contraction and scar
formation
Measure C
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See Figure 4.1 in [Yannas].
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See Figure 4.1 in [Yannas].
Kinetics of change in C
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See Figure 4.2 in [Yannas].
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See Figure 4.2 in [Yannas].
Kinetics of change in C
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See Figure 4.3 in [Yannas].
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See Figure 4.3 in [Yannas].
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See Figure 4.7 in [Yannas].
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See Figure 4.7 in [Yannas].
Measure S (qualitative
assay)
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See Table 4.1 in [Yannas].
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e 4.1 in [Yannas].
2. Synthesis of an epidermis
Structure. Five cell layers (strata); 100 μm
thick. Basal layer is closest to
BM…stratum corneum is farthest out. Cell
maturation gradient (increasing keratin
content away from BM). Tissue turns over
every 25-50 days.
Function. Protection against dehydration
and microorganisms (primarily stratum
corneum). Also protection against
mechanical, thermal, chemical, UV insults.
Schematic
view of
epidermis
2. Synthesis of an epidermis [Cont.]
Synthesis in vitro. Epidermis ? trypsinization
? dissociated keratinocytes (KC).
Condensation of KC to epidermis requires
nondiffusible substrate (e.g., plastic surface)
but not growth factors or dermal substrate.
In vivo. Epidermis synthesized spontaneously
by KC, originally at the defect edge. KC
dissociate spontaneously, migrate over
residual dermis toward “center” of defect,
synthesize BM and reform epidermis.
2B. Synthesis of BM
Structure. BM structure similar in all organs.
100 nm thick. Egg-carton topology in skin.
Layer closest to epidermis is 20-40 nm
thick (lamina lucida; mostly laminin).
Intermediate layer 40-50 nm thick (lamina
densa; type IV collagen). Next to stroma is
fibroreticular layer (anchoring fibrils
based on type VII collagen) that connects
with type I collagen fibers in dermis via
anchoring plaques. Hemidesmosomes
connect basal cells to BM (tonofilaments).
injury mode
(blister)
through epidermis:
reversible healing
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See Figure 2.6 in [Yannas].
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See Figure 2.6 in [Yannas].
between epidermis
and dermis:
reversible healing
through dermis:
irreversible healing
Skin
basement
membrane
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See Figure 5.1 in [Yannas].
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See Figure 5.1 in [Yannas].
LL, lamina
lucida
LD, lamina
densa
d, dermis
2B. Synthesis of BM [Cont.]
Function. Boundary restricting transfer of
cells and molecules; anchorage matrix for
epithelial cells; mechanically competent
“adhesive” layer binding epithelia to
stroma; possibly “scaffold” facilitating
repair after injury.
2B. Synthesis of BM [Cont.]
Synthesis in vitro. KC cultures in serum-free
medium are transferred to solid surface.
BM minus anchoring fibrils is synthesized.
In vivo. KC sheets are grafted on dermis-free
defect; synthesize BM minus anchoring
fibrils. Complete BM formed when
cultured KC sheets are grafted on dermis.
Table 5.1
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See Table 5.1 in [Yannas].
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See Table 5.1 in [Yannas].
2B. Synthesis of BM [Cont.]
Mechanical failure of dermal-epidermal junction.
-- 1952-56 Billingham et al. Epidermal sheets or
KC suspensions grafted on dermis-free
surface failed to adhere (“avulsion”).
-- 1977 Rheinwald and Green (RG) achieved KC
culture expansion to KC sheets by 10,000X in
3 weeks.
-- 1980-95 Clinical studies of KC sheets prepared
by RG method were terminated after
completing 105 of them. Problem: avulsion of
KC sheets from muscle substrate.
-- 1988-95 Woodley, Grinnell, Carver, Cooper et
al. identified source of failure: lack of
integration of BM to muscle substrate.
3. Synthesis of dermis
Structure. Consists of two layers: Papillary
dermis just below epidermis, comprising
loosely packed, thin, type I collagen fibers,
as well as dermal papillae with vascular
loops and nerve endings. Reticular dermis
comprises closely packed, thicker, type I
collagen fibers; also elastin fibers.
Mechanically robust tissue comprises two
interpenetrating networks of stiff, crystalline
collagen fibers and extensible, amorphous,
elastin fibers.
3. Synthesis of dermis (Cont.)
Function. Supports epidermis.
-- tough base absorbs mechanical forces.
-- rich vascular network supports metabolically the
avascular dermis.
-- thermoregulatory control for organism (sweat
glands).
-- tactile, pain, hot/cold sensation, “love” nerve
sensations.
3. Synthesis of dermis (Cont.)
Synthesis in vitro. Not observed.
Synthesis in vivo via sequential synthesis of
dermis and epidermis. Graft biologically
active ECM analog (dermis regeneration
template, DRT) on muscle substrate to
synthesize dermis. Later, KC from defect
margin migrate inside defect and
synthesize BM and epidermis. (also via
simultaneous synthesis---see below)
4. Partial synthesis of skin
Structure. Largest organ (about 18% body
weight). Epidermis bonded to dermis with
“rete ridges” (egg-carton topology).
Elderly lack rete ridges; their skin peels
off easier; mechanical and metabolic role
of rete ridges; not present in scar).
Function. 1. Prevents dehydration and
invasion of bacteria and viruses. 2.
Largest sensory organ, contains receptors
for touch, pressure, pain, temperature. 3.
Helps thermoregulate body (controls heat
transfer). 4. Major source of vitamin D
supply.
4. Partial synthesis of skin [Cont.]
Simultaneous synthesis of epidermis and
dermis.
-- Uncultured KC seeded into DRT and
grafted onto muscle substrate.
-- Contraction arrested and defect perimeter
increased.
-- New tissue inside perimeter analyzed for
skin (no hair).
Evidence for partial synthesis of skin. Table
5.2
functional
properties
of skinTable 5.2
Compare
normal skin,
scar and
regenerated
skin (guinea
pig)
epidermis
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See Table 5.2 in [Yannas].
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e 5.2 in [Yannas].
basement
membrane
dermis
appendages
Scaffold
seeded
with epithelial
cells
KINETICS
OF SKIN
SYNTHESIS
I.
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Scaffold
slowly
degrading
Butler et al., 1998
KINETICS
OF SKIN
SYNTHESIS
II.
Scaffold
degraded;
diffuses
away
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Butler et al., 1998
normal skin
(guinea pig)
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See Figure 5.4 in [Yannas].
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See Figure 5.4 in [Yannas].
scar
v, blood vessels
(absent in scar)
d, dermis
regenerated
skin
rete ridges with
capillary loops
and vascular plexus
underneath
(normal skin)
Image removed due to copyright considerations.
See Figure 5.2 (top left) in [Yannas].
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See Figure 5.2 (top left) in [Yannas].
Verify basement membrane. I: Immunostaining:
Factor VIII for capillary loops
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75 μm
Compton et al., 2000
Verify basement membrane. II. Immunostaining:
α
6
β
4
Integrin for hemidesmosomes
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100 μ m
Compton et al., 2000
Verify basement membrane. III.
Immunostaining: Collagen VII for anchoring
fibrils
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150 μ m
Compton et al., 2000
Regenerated dermis
polarized light
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See Figure 5.3 in [Yannas].
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See Figure 5.3 in [Yannas].
natural light
4. Partial synthesis of skin [Cont.]
In vitro-to-in vivo synthetic routes
-- “Composite graft”. KC- and FB-seeded
DRT cultured in vitro and form epidermis,
before grafting.
-- In another version, use synthetic
polymeric mesh instead of DRT (“living
dermal replacement”).
-- FB are cultured inside collagen gel, then
KC are seeded, before grafting (“living
skin equivalent”).
In vitro or in vivo? Skin
synthesis
Image removed due to copyright considerations.
See Figure 7.1 in [Yannas].
Image removed due to copyright consi erations.
See Figure 7.1 in [Yannas].
5. Comparative regenerative ability
of reactants
See Table 5.3.
Growth factors had no effect on final
configuration (use defect closure rule).
Pharmacological agents, including steroids,
had no effect.
KC sheets were ineffective.
Scaffolds, whether seeded or unseeded with
cells (KC and/or FB), were very effective in
suppressing contraction and scar
synthesis, and inducing regeneration.
Table 5.3
Configuration
of the final
state (skin)
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See Table 5.3 in [Yannas].
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e 5.3 in [Yannas].
Summary
--- Each tissue in skin has been synthesized.
--- Partial synthesis of skin has been also achieved.
--- Reactants added included KC, FB and scaffolds.
--- Epidermis and BM were synthesized in vitro (as
well as in vivo) whereas dermis with rete ridges
was synthesized only in vivo.
Questions to be answered:
Which are the minimal reactants?
What is the difference in conditions between in vitro
and in vivo?